基礎生物学研究所
Matsuda T., Hiyama TY, Niimuara F, Matsusaka T, Fukamizu A, Kobayashi K, Kobayashi K and Noda M. Distinct neural mechanisms for the control of thirst ans salt appetite in the subfornical organ. Nature Neurosci. 20, 230-241, 2017.
Almuriekhi, M., Shintani, T., Fahiminiya, S., Fujikawa, A., Kuboyama, K., Takeuchi, Y., Nawaz, Z., Nadaf, J., Kamel, H., Kitam, A.K., Samiha, Z., Mahmoud, L., Ben-Omran, T., Majewski, J., and Noda, M. (2015). Loss-of-function mutation in APC2 causes Sotos Syndrome features. Cell Reports 10, 1585-1598.
Hiyama, T.Y., Yoshida, M., Matsumoto, M., Suzuki, R., Matsuda, T., Watanabe, E., and Noda, M. (2013). Endothelin-3 expression in the subfornical organ enhances the sensitivity of Nax, the brain sodium-level sensor, to suppress salt intake. Cell Metab. 17, 507-519.
Hiyama, T.Y., Matsuda, S., Fujikawa, A., Matsumoto, M., Watanabe, E., Kajiwara, H., Niimura, F., and Noda, M. (2010). Autoimmunity to the sodium-level sensor in the brain causes essential hypernatremia. Neuron 66, 508-522.
Shimizu, H., Watanabe, E., Hiyama, T.Y., Nagakura, A., Fujikawa, A., Okado, H., Yanagawa, Y., Obata, K., and Noda, M. (2007). Glial Nax channels control lactate signaling to neurons for brain [Na+] sensing. Neuron 54, 59-72.
Shintani, T., Ihara, M., Sakuta, H., Takahashi, H., Watakabe, I., and Noda, M. (2006). Eph receptors are negatively controlled by protein tyrosine phosphatase receptor type O. Nature Neurosci. 9, 761-769.
Fujikawa, A., Shirasaka, D., Yamamoto, S., Ota, H., Yahiro, K., Fukada, M., Shintani, T., Wada, A., Aoyama, N., Hirayama, T., Fukamachi, H., and Noda, M. (2003). Mice deficient in protein tyrosine phosphatase receptor type Z are resistant to gastric ulcer induction by VacA of Helicobacter pylori. Nature Gen. 33, 375-381.