1. DNA damage-dependent acetylation and ubiquitination of H2AX enhances chromatin dynamics .
Ikura, T., Tashiro, S., Kakino, A., Shima, H., Jacob, N., Amunugama, R., Yoder, K., Izumi, S., Kuraoka, I., Tanaka, K., Kimura, H., Ikura, M., Nishikubo, S., Ito, T., Muto, A., Miyagawa, K., Takeda, S., Fishel, R., Igarashi, K., and Kamiya, K
Mol Cell Biol. (in press)

2. Nuclear positioning of the BACH2 gene in BCR-ABL positive leukemia cells.
Ono, A., Kono, K., Ikebe, D., Muto, A., Sun, J., Kobayashi, M., Ueda, K., Melo, J. V., Igarashi, K., Tashiro, S.
Genes, Chromosomes & Cancer 46, 67-74 (2007)

3. Co-repressor SMRT and class II histone deacetylases promote Bach2 nuclear retention and formation of nuclear foci that are responsible for local transcriptional repression.
Hoshino, H., Nishino, T.G., Tashiro, S., Miyazaki, M., Igarashi, K., Horinouchi, S., and Yoshida, M.
J Biochem (Tokyo). 141(5):719-27. (2007)

4. The transcriptional programme of antibody class switching involves the repressor Bach2. Muto A, Tashiro S, Nakajima O, Hoshino H, Takahashi S, Sakoda E, Ikebe D, Yamamoto M, Igarashi K.
Nature. 429(6991):566-71, (2004).

5. Repression of PML nuclear body-associated transcription by oxidative stress-activated Bach2. Tashiro S, Muto A, Tanimoto K, Tsuchiya H, Suzuki H, Hoshino H, Yoshida M, Walter J, Igarashi K.
Mol Cell Biol. 24(8):3473-84, (2004).

6. XRCC3 deficiency results in a defect in recombination and increased endoreduplication in human cells. Yoshihara T, Ishida M, Kinomura A, Katsura M, Tsuruga T, Tashiro S, Asahara T, Miyagawa K.
EMBO J.23(3):670-80, (2004).